Alexander disease mutations differentially sensitize glial fibrillary acidic protein (GFAP) to posttranslational modifications and network disruption by oxidants.
A correction to this paper has been published: the notice, 42150925, from Europe PMC.
Overview
- Department of Molecular and Cellular and Biosciences, Centro de Investigaciones Biológicas Margarita Salas, C.S.I.C., Madrid, 28040, Spain
- Department of Biomedical Sciences, Panum Institute, University of Copenhagen, Copenhagen, 2200, Denmark
Abstract
The abstract is not reproduced here: the paper's license (CC BY-NC-ND) does not allow it. Read it in the paper, at the publisher or on Europe PMC.
Code
The paper links to its data, not to its authors' code: see the Data section.
The paper's code and data availability statement is in the Data section.
Tracing map
A tracing map links a paper to the code its authors published: this paper has none, so it has no map.
Data
Datasets cited
- uniprot.org/
uniprotkb/ , at UniProt; found in the text, “Sample analysis by liquid chromatography-mass…”https:
Availability statements
The paper has a data availability statement and a code and data availability statement. Its license (CC BY-NC-ND) does not allow reproducing them here; in short, from what the harvester recognized in them:
- no repository, dataset or request procedure was recognized in them
Read them in the paper: doi.org/10.1016/j.redox.2026.104103.
Versions
The history of this record: each version stored by the harvester or made by a correction of its authors or of the maintainers of its code, and what changed in its facts. The texts of the paper (its abstract, its availability statements) are not part of it; versions that changed only those are not listed.
Version 1, 30 September 2026: the first record
Recorded: type, language, journal, volume, pages, dates, 8 authors, 8 keywords, 8 MeSH terms, 4 funders, 48 references, 1 integrity notice.
Cite
This paper
Goya-Iglesias, N., Hägglund, P., Martínez-Cenalmor, P., Nybo, T., Lorentzen, L. G., Pajares, M. A., Davies, M. J., & Pérez-Sala, D. (2026). Alexander disease mutations differentially sensitize glial fibrillary acidic protein (GFAP) to posttranslational modifications and network disruption by oxidants. Redox biology, 92, 104103. https://
BibTeX
@article{goyaiglesias202
author = {Goya-Iglesias, Nuria and Hägglund, Per and Martínez-Cenalmor, Paula and Nybo, Tina and Lorentzen, Lasse G and Pajares, María A and Davies, Michael J and Pérez-Sala, Dolores},
title = {{Alexander disease mutations differentially sensitize glial fibrillary acidic protein (GFAP) to posttranslational modifications and network disruption by oxidants}},
journal = {Redox biology},
year = {2026},
month = mar,
volume = {92},
pages = {104103},
publisher = {Elsevier},
issn = {2213-2317},
doi = {10.1016/
url = {https://
pmid = {41830670},
pmcid = {PMC12996815}
}
RIS
TY - JOUR
AU - Goya-Iglesias, Nuria
AU - Hägglund, Per
AU - Martínez-Cenalmor, Paula
AU - Nybo, Tina
AU - Lorentzen, Lasse G
AU - Pajares, María A
AU - Davies, Michael J
AU - Pérez-Sala, Dolores
TI - Alexander disease mutations differentially sensitize glial fibrillary acidic protein (GFAP) to posttranslational modifications and network disruption by oxidants
T2 - Redox biology
J2 - Redox Biol
PY - 2026
DA - 2026/
VL - 92
SP - 104103
SN - 2213-2317
PB - Elsevier
DO - 10.1016/
UR - https://
LA - en
ER -
CSL-JSON
{
"id": "10.1016/
"type": "article-journal",
"title": "Alexander disease mutations differentially sensitize glial fibrillary acidic protein (GFAP) to posttranslational modifications and network disruption by oxidants",
"container-title": "Redox biology",
"author": [
{
"family": "Goya-Iglesias",
"given": "Nuria"
},
{
"family": "Hägglund",
"given": "Per"
},
{
"family": "Martínez-Cenalmor",
"given": "Paula"
},
{
"family": "Nybo",
"given": "Tina"
},
{
"family": "Lorentzen",
"given": "Lasse G"
},
{
"family": "Pajares",
"given": "María A"
},
{
"family": "Davies",
"given": "Michael J"
},
{
"family": "Pérez-Sala",
"given": "Dolores"
}
],
"container-title-short":
"volume": "92",
"page": "104103",
"DOI": "10.1016/
"PMID": "41830670",
"PMCID": "PMC12996815",
"ISSN": "2213-2317",
"publisher": "Elsevier",
"URL": "https://
"language": "en",
"issued": {
"date-parts": [
[
2026,
3,
4
]
]
}
}
Similar papers
The papers with a page that share the most with this one: the tools found in their code, their categories, datasets, cited references and authors, the rarest counting most.
- [1] doi:10.1186/s40478-026-02240-y
- GFAP degradation in TBI: linking novel modified products to astrocyte pathology and patient outcome.Journal: Acta neuropathologica communicationsIn common: other, 4 references
- [2] doi:10.1016/j.jbc.2026.113363
- The Src family kinase YES regulates DNAJB6b chaperone activity to modulate tau aggregation in Alzheimer's disease.Journal: The Journal of biological chemistryIn common: uniprot.org/uniprotkb/https:, cellular / molecular
- [3] doi:10.1016/j.isci.2026.116097
- Sexual identity of Akh neurons shapes Nucb1-dependent metabolic and reproductive plasticity.Journal: iScienceIn common: uniprot.org/uniprotkb/https:, cellular / molecular
- [4] doi:10.1016/j.isci.2026.115554
- Computationally guided discovery of Ly6e/
LY6E-dependent AAV capsid variants. Journal: iScienceIn common: uniprot.org/uniprotkb/https:, cellular / molecular - [5] doi:10.1177/11769351261460625
- GFAP-Dependent Transcriptional Dynamics and Cellular Heterogeneity in Primary, Recurrent, and Grade III Gliomas.Journal: Cancer informaticsIn common: other, genetics / omics, cellular / molecular, 1 reference
- [6] doi:10.1016/j.isci.2026.116439 [code]
- Decoding the role of transcriptomic clocks in the human prefrontal cortex.Journal: iScienceIn common: genetics / omics, 1 reference
- [7] doi:10.1073/pnas.2605750123
- Light- and temperature-sensitive seizures are regulated by spatially distinct cortex glial populations in the central nervous system.Journal: Proceedings of the National Academy of Sciences of the United States of AmericaIn common: cellular / molecular, 1 reference
Contribute
The authors of this paper can claim it, correct its record and validate its tracing map, and the maintainers of its code (its owner, or a public member of its organization) correct what it says of their repository; anyone signed in can ask for its removal. Every request goes to OSCR's own machine, which answers it; your account page follows them.
Sign in with ORCID to claim this paper as one of its authors, correct its record or validate its tracing map: when the paper's metadata lists your ORCID iD, you are recognized at once. Maintainers of its code: sign in with GitHub, then claim the repository on your account page.
Claim this paper
Correct its record
Say what each link of this record is, remove the ones that are not the paper's, add the ones that are missing. The correction becomes a new version of the record, in its Versions section.
Request its removal
To ask OSCR to remove this record, the copies of its authors' scripts or its tracing map, use the removal request page: signed in, you say who you are, what to remove and why, then review and confirm the request. Published rules decide every request (how).
Discussion, reproductions, activity
Discussion: questions and error reports about this paper and its code, from signed-in readers and its authors. It opens with sign-in.
Reproductions: reports from readers who ran the authors' code: what they reproduced, with which environment, commit and data. It opens with sign-in.
Activity: what happens around this paper: new versions of its record, its map's validation, discussions and reproductions. It opens with sign-in.
