Oligodendrocyte precursor cells-microglia crosstalk via BMP4 drives microglial neuroprotective response and mitigates Alzheimer's disease.
Overview
Abstract
The abstract is not reproduced here: the paper's license (CC BY-NC-ND) does not allow it. Read it in the paper, at the publisher or on Europe PMC.
Code
The paper says that its authors' code is available on request: it was not published with the paper, so there is nothing to verify.
The paper's code and data availability statement is in the Data section.
Tracing map
A tracing map links a paper to the code its authors published: this paper has none (its code is available on request), so it has no map.
Data
Data links
- ncbi.nlm.nih.gov/
geo , NCBI; found in “Data availability”
Code and data availability statement
The paper has a code and data availability statement. Its license (CC BY-NC-ND) does not allow reproducing it here; in short, from what the harvester recognized in it:
- it points to a dataset: NCBI
- it says that the code is available on request
Read it in the paper: doi.org/10.1038/s41392-026-02620-9.
Versions
The history of this record: each version stored by the harvester or made by a correction of its authors or of the maintainers of its code, and what changed in its facts. The texts of the paper (its abstract, its availability statements) are not part of it; versions that changed only those are not listed.
Version 1, 30 September 2026: the first record
Recorded: type, language, journal, volume, issue, pages, dates, 5 authors, 2 keywords, 11 MeSH terms, 1 funder, 67 references.
Cite
This paper
Baek, S., Jang, J., Yeo, S., Jung, H. J., & Choe, Y. (2026). Oligodendrocyte precursor cells-microglia crosstalk via BMP4 drives microglial neuroprotective response and mitigates Alzheimer's disease. Signal transduction and targeted therapy, 11(1), 109. https://
BibTeX
@article{baek2026oligode
author = {Baek, Soonbong and Jang, Jaemyung and Yeo, Seungeun and Jung, Hyun Jin and Choe, Youngshik},
title = {{Oligodendrocyte precursor cells-microglia crosstalk via BMP4 drives microglial neuroprotective response and mitigates Alzheimer's disease}},
journal = {Signal transduction and targeted therapy},
year = {2026},
month = mar,
volume = {11},
number = {1},
pages = {109},
publisher = {Nature Publishing Group},
issn = {2095-9907},
doi = {10.1038/
url = {https://
pmid = {41881962},
pmcid = {PMC13018198}
}
RIS
TY - JOUR
AU - Baek, Soonbong
AU - Jang, Jaemyung
AU - Yeo, Seungeun
AU - Jung, Hyun Jin
AU - Choe, Youngshik
TI - Oligodendrocyte precursor cells-microglia crosstalk via BMP4 drives microglial neuroprotective response and mitigates Alzheimer's disease
T2 - Signal transduction and targeted therapy
J2 - Signal Transduct Target Ther
PY - 2026
DA - 2026/
VL - 11
IS - 1
SP - 109
SN - 2095-9907
PB - Nature Publishing Group
DO - 10.1038/
UR - https://
LA - en
ER -
CSL-JSON
{
"id": "10.1038/
"type": "article-journal",
"title": "Oligodendrocyte precursor cells-microglia crosstalk via BMP4 drives microglial neuroprotective response and mitigates Alzheimer's disease",
"container-title": "Signal transduction and targeted therapy",
"author": [
{
"family": "Baek",
"given": "Soonbong"
},
{
"family": "Jang",
"given": "Jaemyung"
},
{
"family": "Yeo",
"given": "Seungeun"
},
{
"family": "Jung",
"given": "Hyun Jin"
},
{
"family": "Choe",
"given": "Youngshik"
}
],
"container-title-short":
"volume": "11",
"issue": "1",
"page": "109",
"DOI": "10.1038/
"PMID": "41881962",
"PMCID": "PMC13018198",
"ISSN": "2095-9907",
"publisher": "Nature Publishing Group",
"URL": "https://
"language": "en",
"issued": {
"date-parts": [
[
2026,
3,
25
]
]
}
}
Similar papers
The papers with a page that share the most with this one: the tools found in their code, their categories, datasets, cited references and authors, the rarest counting most.
- [1] doi:10.1186/s40478-023-01637-3 [code]
- Abnormal accumulation of extracellular vesicles in hippocampal dystrophic axons and regulation by the primary cilia in Alzheimer's disease.Journal: Acta neuropathologica communicationsIn common: Alzheimer's / dementia, mouse, cellular / molecular, 2 authors
- [2] doi:10.1186/s13024-024-00793-x
- APOE Christchurch enhances a disease-associated microglial response to plaque but suppresses response to tau pathology.Journal: Molecular neurodegenerationIn common: Alzheimer's / dementia, mouse, cellular / molecular, 4 references
- [3] doi:10.3389/fmolb.2023.1250335 [code]
- Primary cilia-mediated regulation of microglial secretion in Alzheimer's disease.Journal: Frontiers in molecular biosciencesIn common: Alzheimer's / dementia, mouse, cellular / molecular, author Jaemyung Jang
- [4] doi:10.1007/s00424-023-02837-5
- Oligodendrocyte precursor cells: the multitaskers in the brain.Journal: Pflugers Archiv : European journal of physiologyIn common: cellular / molecular, 4 references
- [5] doi:10.1186/s13024-024-00759-z
- Contribution of amyloid deposition from oligodendrocytes in a mouse model of Alzheimer's disease.Journal: Molecular neurodegenerationIn common: Alzheimer's / dementia, mouse, cellular / molecular, 3 references
- [6] doi:10.1038/s41467-025-66222-9
- Sulfatide deficiency-induced astrogliosis and myelin lipid dyshomeostasis are independent of TREM2-mediated microglial activation.Journal: Nature communicationsIn common: Alzheimer's / dementia, mouse, cellular / molecular, 3 references
- [7] doi:10.1002/glia.70210 [code]
- Predicting Relevant Microglia-Associated Cell-Cell Communication Pathways in Alzheimer's Disease: A Role for SPP1.Journal: GliaIn common: Alzheimer's / dementia, cellular / molecular, 3 references
- [8] doi:10.1038/s41467-025-67930-y
- Chromatin remodeling factor BAF155 coordinates oligodendroglial-neurona
l communications linked to regional myelination and autism-like behavioral deficits in mice. Journal: Nature communicationsIn common: mouse, cellular / molecular, 3 references - [9] doi:10.1186/s12974-024-03307-0 [code]
- Microglia in the aged brain develop a hypoactive molecular phenotype after surgery.Journal: Journal of neuroinflammationIn common: mouse, cellular / molecular, 3 references
- [10] doi:10.1007/s12035-025-04997-0
- Decoding Alzheimer's Disease: Single-Cell Sequencing Uncovers Brain Cell Heterogeneity and Pathogenesis.Journal: Molecular neurobiologyIn common: Alzheimer's / dementia, cellular / molecular, 3 references
Contribute
The authors of this paper can claim it, correct its record and validate its tracing map, and the maintainers of its code (its owner, or a public member of its organization) correct what it says of their repository; anyone signed in can ask for its removal. Every request goes to OSCR's own machine, which answers it; your account page follows them.
Sign in with ORCID to claim this paper as one of its authors, correct its record or validate its tracing map: when the paper's metadata lists your ORCID iD, you are recognized at once. Maintainers of its code: sign in with GitHub, then claim the repository on your account page.
Claim this paper
Correct its record
Say what each link of this record is, remove the ones that are not the paper's, add the ones that are missing. The correction becomes a new version of the record, in its Versions section.
Request its removal
To ask OSCR to remove this record, the copies of its authors' scripts or its tracing map, use the removal request page: signed in, you say who you are, what to remove and why, then review and confirm the request. Published rules decide every request (how).
Discussion, reproductions, activity
Discussion: code errors and mismatches between the code and the paper about this paper and its code, from readers and its authors, beside the paper and its tracing map.
Reproductions: reports from readers who ran the authors' code and did not get the paper's result, what they ran, with which environment, commit and data.
Activity: what happens around this paper: new versions of its record, its map's validation, discussions and reproductions. It opens with sign-in.