MEG3 in glioma stem cells promotes glioblastoma angiogenesis through FUBP3-mediated VGF expression.
Overview
- Department of Pathology, the First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, Anhui 230036 China
- Intelligent Pathology Institute, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, Anhui 230036 China
- Department of Pathology, Centre for Leading Medicine and Advanced Technologies of IHM, The First Affiliated Hospital of USTC, Division of Life Sciences and Medicine, University of Science and Technology of China, Hefei, Anhui 230036 China
Abstract
The abstract is not reproduced here: the paper's license (CC BY-NC-ND) does not allow it. Read it in the paper, at the publisher or on Europe PMC.
Code
The paper links to its data and to no code of its authors: see the Data section.
Tracing map
A tracing map links a paper to the code its authors published: this paper has none, so it has no map.
Data
Datasets cited
- geo:GSE194329, at NCBI GEO; found in the text, “Spatial Transcriptomic Data Analysis”
Data availability statement
The paper has a data availability statement. Its license (CC BY-NC-ND) does not allow reproducing it here; in short, from what the harvester recognized in it:
- no repository, dataset or request procedure was recognized in it
Read it in the paper: doi.org/10.1007/s10238-026-02097-4.
Versions
The history of this record: each version stored by the harvester or made by a correction of its authors or of the maintainers of its code, and what changed in its facts. The texts of the paper (its abstract, its availability statements) are not part of it; versions that changed only those are not listed.
Version 1, 30 September 2026: the first record
Recorded: type, language, journal, volume, issue, pages, dates, 7 authors, 5 keywords, 15 MeSH terms, 3 funders, 33 references.
Cite
This paper
Li, Y., Li, Y., Tang, J., Kong, X., Sun, S., Wang, W., & Wu, H. (2026). MEG3 in glioma stem cells promotes glioblastoma angiogenesis through FUBP3-mediated VGF expression. Clinical and experimental medicine, 26(1), 208. https://
BibTeX
@article{li2026meg3,
author = {Li, Yuan and Li, Yujie and Tang, Jiaxin and Kong, Xue and Sun, Sibai and Wang, Wei and Wu, Haibo},
title = {{MEG3 in glioma stem cells promotes glioblastoma angiogenesis through FUBP3-mediated VGF expression}},
journal = {Clinical and experimental medicine},
year = {2026},
month = mar,
volume = {26},
number = {1},
pages = {208},
publisher = {Springer Science+Business Media},
issn = {1591-8890},
doi = {10.1007/
url = {https://
pmid = {41866561},
pmcid = {PMC13046654}
}
RIS
TY - JOUR
AU - Li, Yuan
AU - Li, Yujie
AU - Tang, Jiaxin
AU - Kong, Xue
AU - Sun, Sibai
AU - Wang, Wei
AU - Wu, Haibo
TI - MEG3 in glioma stem cells promotes glioblastoma angiogenesis through FUBP3-mediated VGF expression
T2 - Clinical and experimental medicine
J2 - Clin Exp Med
PY - 2026
DA - 2026/
VL - 26
IS - 1
SP - 208
SN - 1591-8890
PB - Springer Science+Business Media
DO - 10.1007/
UR - https://
LA - en
ER -
CSL-JSON
{
"id": "10.1007/
"type": "article-journal",
"title": "MEG3 in glioma stem cells promotes glioblastoma angiogenesis through FUBP3-mediated VGF expression",
"container-title": "Clinical and experimental medicine",
"author": [
{
"family": "Li",
"given": "Yuan"
},
{
"family": "Li",
"given": "Yujie"
},
{
"family": "Tang",
"given": "Jiaxin"
},
{
"family": "Kong",
"given": "Xue"
},
{
"family": "Sun",
"given": "Sibai"
},
{
"family": "Wang",
"given": "Wei"
},
{
"family": "Wu",
"given": "Haibo"
}
],
"container-title-short":
"volume": "26",
"issue": "1",
"page": "208",
"DOI": "10.1007/
"PMID": "41866561",
"PMCID": "PMC13046654",
"ISSN": "1591-8890",
"publisher": "Springer Science+Business Media",
"URL": "https://
"language": "en",
"issued": {
"date-parts": [
[
2026,
3,
23
]
]
}
}
Similar papers
The papers with a page that share the most with this one: the tools found in their code, their categories, datasets, cited references and authors, the rarest counting most.
- [1] doi:10.1186/s40478-025-02036-6
- Single-cell transcriptome sequencing reveals new epithelial-stromal associated mesenchymal-like subsets in recurrent gliomas.Journal: Acta neuropathologica communicationsIn common: NCBI GEO GSE194329, genetics / omics, other condition, mouse, 1 other category
- [2] doi:10.1111/cns.14822
- Evidences of neurological injury caused by COVID-19 from glioma tissues and glioma organoids.Journal: CNS neuroscience & therapeuticsIn common: NCBI GEO GSE194329, genetics / omics, other condition
- [3] doi:10.1002/cam4.70288
- New Anti-Angiogenic Therapy for Glioblastoma With the Anti-Depressant Sertraline.Journal: Cancer medicineIn common: other condition, mouse, 3 references
- [4] doi:10.1186/s12967-026-08397-3
- FAM135B suppresses glioblastoma angiogenesis via stabilizing the IKK complex and inactivating the NF-κB/
IL-6 signaling pathway. Journal: Journal of translational medicineIn common: other condition, mouse, cellular / molecular, 3 references - [5] doi:10.1038/s12276-026-01801-4 [code]
- Putative glioblastoma origin-like cells in the subventricular zone: isolation and characterization.Journal: Experimental & molecular medicineIn common: genetics / omics, other condition, mouse, 1 other category, 2 references
- [6] doi:10.1038/s41586-024-07493-y [code]
- Single-cell atlas of the human brain vasculature across development, adulthood and disease.Journal: NatureIn common: genetics / omics, other condition, 2 references
- [7] doi:10.1038/s41417-022-00582-y
- Single-cell RNA sequencing reveals changes in glioma-associated macrophage polarization and cellular states of malignant gliomas with high AQP4 expression.Journal: Cancer gene therapyIn common: genetics / omics, other condition, cellular / molecular, 2 references
- [8] doi:10.1080/10717544.2026.2660007
- Angiopep-2-decorated bacterial outer membrane vesicles penetrate the blood-brain barrier for glioblastoma chemo-immunotherapy.Journal: Drug deliveryIn common: other condition, mouse, cellular / molecular, 2 references
- [9] doi:10.1111/cas.70014
- Abnormal Vessels Potentially Accelerate Glioblastoma Proliferation by Inducing the Protumor Activation of Macrophages.Journal: Cancer scienceIn common: other condition, mouse, cellular / molecular, 2 references
- [10] doi:10.1038/s44321-025-00195-6
- Exploiting metabolic vulnerability in glioblastoma using a brain-penetrant drug with a safe profile.Journal: EMBO molecular medicineIn common: other condition, mouse, cellular / molecular, 2 references
Contribute
The authors of this paper can claim it, correct its record and validate its tracing map, and the maintainers of its code (its owner, or a public member of its organization) correct what it says of their repository; anyone signed in can ask for its removal. Every request goes to OSCR's own machine, which answers it; your account page follows them.
Sign in with ORCID to claim this paper as one of its authors, correct its record or validate its tracing map: when the paper's metadata lists your ORCID iD, you are recognized at once. Maintainers of its code: sign in with GitHub, then claim the repository on your account page.
Claim this paper
Correct its record
Say what each link of this record is, remove the ones that are not the paper's, add the ones that are missing. The correction becomes a new version of the record, in its Versions section.
Request its removal
To ask OSCR to remove this record, the copies of its authors' scripts or its tracing map, use the removal request page: signed in, you say who you are, what to remove and why, then review and confirm the request. Published rules decide every request (how).
Discussion, reproductions, activity
Discussion: code errors and mismatches between the code and the paper about this paper and its code, from readers and its authors, beside the paper and its tracing map.
Reproductions: reports from readers who ran the authors' code and did not get the paper's result, what they ran, with which environment, commit and data.
Activity: what happens around this paper: new versions of its record, its map's validation, discussions and reproductions. It opens with sign-in.
