Astrocytes mobilize a broader repertoire of lysosomal repair mechanisms than neurons.
Overview
- Department of Neuroscience, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA 19104, USA
- Department of Physiology, Perelman School of Medicine at the University of Pennsylvania, Philadelphia, PA 19104, USA
Abstract
The abstract is not reproduced here: the paper's license (CC BY-NC-ND) does not allow it. Read it in the paper, at the publisher or on Europe PMC.
Code
The paper says that its authors' code is available on request: it was not published with the paper, so there is nothing to verify.
The paper's code and data availability statement is in the Data section.
Tracing map
A tracing map links a paper to the code its authors published: this paper has none (its code is available on request), so it has no map.
Data
No dataset and no data link were found in the paper.
Code and data availability statement
The paper has a code and data availability statement. Its license (CC BY-NC-ND) does not allow reproducing it here; in short, from what the harvester recognized in it:
- it says that the data are available on request
- it says that the code is available on request
Read it in the paper: doi.org/10.1016/j.isci.2026.116866.
Versions
The history of this record: each version stored by the harvester or made by a correction of its authors or of the maintainers of its code, and what changed in its facts. The texts of the paper (its abstract, its availability statements) are not part of it; versions that changed only those are not listed.
Version 1, 27 September 2026: the first record
Recorded: type, language, journal, volume, issue, pages, dates, 3 authors, 8 keywords, 2 funders, 126 references, 46 RRIDs.
Cite
This paper
Smith, E. M., Chanaday, N. L., & Maday, S. (2026). Astrocytes mobilize a broader repertoire of lysosomal repair mechanisms than neurons. iScience, 29(8), 116866. https://
BibTeX
@article{smith2026astroc
author = {Smith, Erin M. and Chanaday, Natali L. and Maday, Sandra},
title = {{Astrocytes mobilize a broader repertoire of lysosomal repair mechanisms than neurons}},
journal = {iScience},
year = {2026},
month = aug,
volume = {29},
number = {8},
pages = {116866},
publisher = {Elsevier},
issn = {2589-0042},
doi = {10.1016/
url = {https://
pmid = {42620891},
pmcid = {PMC13487023}
}
RIS
TY - JOUR
AU - Smith, Erin M.
AU - Chanaday, Natali L.
AU - Maday, Sandra
TI - Astrocytes mobilize a broader repertoire of lysosomal repair mechanisms than neurons
T2 - iScience
J2 - iScience
PY - 2026
DA - 2026/
VL - 29
IS - 8
SP - 116866
SN - 2589-0042
PB - Elsevier
DO - 10.1016/
UR - https://
LA - en
ER -
CSL-JSON
{
"id": "10.1016/
"type": "article-journal",
"title": "Astrocytes mobilize a broader repertoire of lysosomal repair mechanisms than neurons",
"container-title": "iScience",
"author": [
{
"family": "Smith",
"given": "Erin M."
},
{
"family": "Chanaday",
"given": "Natali L."
},
{
"family": "Maday",
"given": "Sandra"
}
],
"container-title-short":
"volume": "29",
"issue": "8",
"page": "116866",
"DOI": "10.1016/
"PMID": "42620891",
"PMCID": "PMC13487023",
"ISSN": "2589-0042",
"publisher": "Elsevier",
"URL": "https://
"language": "en",
"issued": {
"date-parts": [
[
2026,
8,
12
]
]
}
}
Similar papers
The papers with a page that share the most with this one: the tools found in their code, their categories, datasets, cited references and authors, the rarest counting most.
- [1] doi:10.1371/journal.pone.0345890
- Differential regulation of p62-ubiquitin conjugates in neurons versus astrocytes during cellular stress.Journal: PloS oneIn common: cellular / molecular, 13 references
- [2] doi:10.1038/s44318-026-00817-w
- The C9orf72/
SMCR8 complex maintains microglial homeostasis via RAB8A-ESCRT-mediated lysosomal repair. Journal: The EMBO journalIn common: cellular / molecular, 10 references - [3] doi:10.3389/fnagi.2026.1787252
- Age-associated neuronal micronuclei formation and transfer to microglia.Journal: Frontiers in aging neuroscienceIn common: cellular / molecular, 3 references
- [4] doi:10.1038/s41467-026-72568-5 [code]
- Endosome maturation is orchestrated by inside-out proton signaling through a Na&
lt;sup& gt;+& lt;/ sup& gt;/ H& lt;sup& gt;+& lt;/ sup& gt; exchanger and pH-dependent Rab GTPase cycling. Journal: Nature communicationsIn common: cellular / molecular, 2 references - [5] doi:10.1038/s41531-026-01382-z
- Preformed fibrils of α-synuclein rapidly activate LRRK2 on early endosomes, driving Rab5 phosphorylation and disrupting endolysosomal and synaptic function.Journal: NPJ Parkinson's diseaseIn common: cellular / molecular, 2 references
- [6] doi:10.1038/s41586-026-10648-8
- Confined migration induces non-lethal DNA damage in developing neurons.Journal: NatureIn common: cellular / molecular, 2 references
- [7] doi:10.1111/jnc.70551 [code]
- Synaptobrevin-2 Containing Extracellular Vesicles Are Rapidly Incorporated Into Mammalian Neurons via a Dynamin-Dependent Pathway.Journal: Journal of neurochemistryIn common: cellular / molecular, 2 references
- [8] doi:10.1016/j.stemcr.2026.103004
- Induction of human pruriceptors from pluripotent stem cells via transcription factors.Journal: Stem cell reportsIn common: cellular / molecular, 2 references
- [9] doi:10.1038/s44318-026-00847-4
- Local autophagy impairment triggers brain-wide presynaptic remodeling and resilience.Journal: The EMBO journalIn common: cellular / molecular, 2 references
- [10] doi:10.1038/s41467-026-73003-5 [code]
- Gpnmb defines a phagocytic state of microglia linked to cell death in prion disease mouse model.Journal: Nature communicationsIn common: cellular / molecular, 2 references
Contribute
The authors of this paper can claim it, correct its record and validate its tracing map, and the maintainers of its code (its owner, or a public member of its organization) correct what it says of their repository; anyone signed in can ask for its removal. Every request goes to OSCR's own machine, which answers it; your account page follows them.
Sign in with ORCID to claim this paper as one of its authors, correct its record or validate its tracing map: when the paper's metadata lists your ORCID iD, you are recognized at once. Maintainers of its code: sign in with GitHub, then claim the repository on your account page.
Claim this paper
Correct its record
Say what each link of this record is, remove the ones that are not the paper's, add the ones that are missing. The correction becomes a new version of the record, in its Versions section.
Request its removal
To ask OSCR to remove this record, the copies of its authors' scripts or its tracing map, use the removal request page: signed in, you say who you are, what to remove and why, then review and confirm the request. Published rules decide every request (how).
Discussion, reproductions, activity
Discussion: questions and error reports about this paper and its code, from signed-in readers and its authors. It opens with sign-in.
Reproductions: reports from readers who ran the authors' code: what they reproduced, with which environment, commit and data. It opens with sign-in.
Activity: what happens around this paper: new versions of its record, its map's validation, discussions and reproductions. It opens with sign-in.