Amyloid-β aggregates induce vasculopathy via ferroptosis in brain endothelial cells.
Overview
- Dementia Research Group, Korea Brain Research Institute, Daegu, Republic of Korea
- Daegu Gyeongbuk Institute of Science and Technology, Daegu, Republic of Korea
- Department of Basic and Clinical Neuroscience, Institute of Psychiatry, Psychology & Neuroscience, King's College London, London, UK
- Ginsentology Research Laboratory and Department of Physiology, College of Veterinary Medicine, Konkuk University, Seoul, Republic of Korea
Abstract
The abstract is not reproduced here: the paper's license (CC BY-NC-ND) does not allow it. Read it in the paper, at the publisher or on Europe PMC.
Code
The paper links to its data, not to its authors' code: see the Data section.
Tracing map
A tracing map links a paper to the code its authors published: this paper has none, so it has no map.
Data
Datasets cited
- geo:GSE310780, at NCBI GEO; found in “DATA AVAILABILITY STATEMENT”
Data availability statement
The paper has a data availability statement. Its license (CC BY-NC-ND) does not allow reproducing it here; in short, from what the harvester recognized in it:
- it points to a dataset: NCBI GEO GSE310780
- it says that the data are available on request
Read it in the paper: doi.org/10.1111/bpa.70074.
Versions
The history of this record: each version stored by the harvester or made by a correction of its authors or of the maintainers of its code, and what changed in its facts. The texts of the paper (its abstract, its availability statements) are not part of it; versions that changed only those are not listed.
Version 1, 30 September 2026: the first record
Recorded: type, language, journal, volume, issue, pages, dates, 8 authors, 5 keywords, 14 MeSH terms, 3 funders, 69 references.
Cite
This paper
Son, S., Kim, S., Jeon, M., Choi, M., Kim, K., Lee, S., Nah, S., & Kim, D. (2026). Amyloid-β aggregates induce vasculopathy via ferroptosis in brain endothelial cells. Brain pathology (Zurich, Switzerland), 36(4), e70074. https://
BibTeX
@article{son2026amyloid,
author = {Son, Suhyeon and Kim, Suji and Jeon, Min‐Tae and Choi, Moonseok and Kim, Kyu‐Sung and Lee, Suji and Nah, Seung‐Yeol and Kim, Do‐Geun},
title = {{Amyloid-β aggregates induce vasculopathy via ferroptosis in brain endothelial cells}},
journal = {Brain pathology (Zurich, Switzerland)},
year = {2026},
month = mar,
volume = {36},
number = {4},
pages = {e70074},
publisher = {Wiley},
issn = {1015-6305},
doi = {10.1111/
url = {https://
pmid = {41765417},
pmcid = {PMC13240017}
}
RIS
TY - JOUR
AU - Son, Suhyeon
AU - Kim, Suji
AU - Jeon, Min‐Tae
AU - Choi, Moonseok
AU - Kim, Kyu‐Sung
AU - Lee, Suji
AU - Nah, Seung‐Yeol
AU - Kim, Do‐Geun
TI - Amyloid-β aggregates induce vasculopathy via ferroptosis in brain endothelial cells
T2 - Brain pathology (Zurich, Switzerland)
J2 - Brain Pathol
PY - 2026
DA - 2026/
VL - 36
IS - 4
SP - e70074
SN - 1015-6305
PB - Wiley
DO - 10.1111/
UR - https://
LA - en
ER -
CSL-JSON
{
"id": "10.1111/
"type": "article-journal",
"title": "Amyloid-β aggregates induce vasculopathy via ferroptosis in brain endothelial cells",
"container-title": "Brain pathology (Zurich, Switzerland)",
"author": [
{
"family": "Son",
"given": "Suhyeon"
},
{
"family": "Kim",
"given": "Suji"
},
{
"family": "Jeon",
"given": "Min‐Tae"
},
{
"family": "Choi",
"given": "Moonseok"
},
{
"family": "Kim",
"given": "Kyu‐Sung"
},
{
"family": "Lee",
"given": "Suji"
},
{
"family": "Nah",
"given": "Seung‐Yeol"
},
{
"family": "Kim",
"given": "Do‐Geun"
}
],
"container-title-short":
"volume": "36",
"issue": "4",
"page": "e70074",
"DOI": "10.1111/
"PMID": "41765417",
"PMCID": "PMC13240017",
"ISSN": "1015-6305",
"publisher": "Wiley",
"URL": "https://
"language": "en",
"issued": {
"date-parts": [
[
2026,
3,
1
]
]
}
}
Similar papers
The papers with a page that share the most with this one: the tools found in their code, their categories, datasets, cited references and authors, the rarest counting most.
- [1] doi:10.1038/s43587-026-01204-0
- Fibronectin mediates APOE4-driven blood-brain barrier dysfunction in Alzheimer's disease.Journal: Nature agingIn common: Alzheimer's / dementia, mouse, cellular / molecular, 3 references
- [2] doi:10.1093/neuonc/noag080
- Laser interstitial thermal therapy enhances bidirectional blood-brain barrier permeability in glioblastoma.Journal: Neuro-oncologyIn common: mouse, 3 references
- [3] doi:10.1016/j.crmeth.2026.101455
- An optimized method to visualize lipid droplets in mouse brain tissue.Journal: Cell reports methodsIn common: mouse, 3 references
- [4] doi:10.1016/j.redox.2026.104155
- Ferroptosis-related mechanisms in prion diseases provide insights into neurodegeneration and reveal therapeutic implications.Journal: Redox biologyIn common: mouse, cellular / molecular, 2 references
- [5] doi:10.3892/mmr.2026.13967
- Sedanolide alleviates LPS‑induced depressive‑like behaviors by modulating the C3a/
C3aR signaling axis and microglial glycolysis. Journal: Molecular medicine reportsIn common: mouse, cellular / molecular, 3 references - [6] doi:10.3390/antiox15050634
- Lipid Remodeling in Mouse SR-B1-Deficient Embryos with Oxidative Stress-Associated Neural Tube Defects.Journal: Antioxidants (Basel, Switzerland)In common: mouse, 2 references
- [7] doi:10.1038/s41467-026-75367-0
- Heterogenous microglial reactivity contrasts with stable vascular transcriptional programs in mouse models of Alzheimer's, CADASIL, and Traumatic Brain Injury.Journal: Nature communicationsIn common: Alzheimer's / dementia, mouse, cellular / molecular, 2 references
- [8] doi:10.1002/alz.71804
- A meta-refined human Alzheimer's disease-associated gene subset shows partial mouse-model pathway correspondence and limited cross-cohort machine-learning transportability.Journal: Alzheimer's & dementia : the journal of the Alzheimer's AssociationIn common: Alzheimer's / dementia, mouse, cellular / molecular, 2 references
- [9] doi:10.1186/s40035-026-00563-3
- Protein kinase CK2α' as a dual modulator of neuroimmune signaling and synaptic dysfunction in tauopathy.Journal: Translational neurodegenerationIn common: Alzheimer's / dementia, mouse, cellular / molecular, 2 references
- [10] doi:10.1016/j.isci.2026.115638 [code]
- Genome-scale metabolic modeling uncovers cell-type specific signatures associated with APOE variants.Journal: iScienceIn common: Alzheimer's / dementia, cellular / molecular, 2 references
Contribute
The authors of this paper can claim it, correct its record and validate its tracing map, and the maintainers of its code (its owner, or a public member of its organization) correct what it says of their repository; anyone signed in can ask for its removal. Every request goes to OSCR's own machine, which answers it; your account page follows them.
Sign in with ORCID to claim this paper as one of its authors, correct its record or validate its tracing map: when the paper's metadata lists your ORCID iD, you are recognized at once. Maintainers of its code: sign in with GitHub, then claim the repository on your account page.
Claim this paper
Correct its record
Say what each link of this record is, remove the ones that are not the paper's, add the ones that are missing. The correction becomes a new version of the record, in its Versions section.
Request its removal
To ask OSCR to remove this record, the copies of its authors' scripts or its tracing map, use the removal request page: signed in, you say who you are, what to remove and why, then review and confirm the request. Published rules decide every request (how).
Discussion, reproductions, activity
Discussion: questions and error reports about this paper and its code, from signed-in readers and its authors. It opens with sign-in.
Reproductions: reports from readers who ran the authors' code: what they reproduced, with which environment, commit and data. It opens with sign-in.
Activity: what happens around this paper: new versions of its record, its map's validation, discussions and reproductions. It opens with sign-in.